Coronary Artery Disease (CAD) is a prevalent cardiovascular condition characterized by the accumulation of plaque within the coronary arteries, the blood vessels responsible for supplying oxygen and nutrients to the heart muscle. This plaque, primarily composed of cholesterol, fat, and other substances, can narrow or block the arteries, restricting blood flow to the heart. As a result, the heart may not receive an adequate oxygen supply, leading to chest pain (angina) or, in severe cases, a heart attack. Several risk factors contribute to the development of CAD, including high blood pressure, high cholesterol levels, smoking, diabetes, and a sedentary lifestyle. Prevention and management strategies often involve lifestyle modifications, such as adopting a healthy diet, regular exercise, and avoiding tobacco, along with medications to control blood pressure and cholesterol levels. In some cases, medical interventions like angioplasty or coronary artery bypass surgery may be necessary to restore proper blood flow to the heart. Early detection and comprehensive care are crucial in addressing Coronary Artery Disease and reducing the risk of complications.
Title : Frequency of residual risk in cardiovascular secondary prevention in patients treated at the Reina Fabiola University Clinic
Agustin Joison, Cordoba Catholic University, Argentina
Title : Revolutionizing heart health through Personalized & Precision Medicine (PPM): Towards personalized cardiovascular prevention, precision cardiac care & rehabilitation via optimizing Cardiac Hospital Networks
Sergey Suchkov, N. D. Zelinskii Institute for Organic Chemistry of the Russian Academy of Sciences, Russian Federation
Title : Antibody-proteases as unique biomarkers, potential targets and translational tools of the next step generation to be used in personalized and precision cardiology practice
Sergey Suchkov, N. D. Zelinskii Institute for Organic Chemistry of the Russian Academy of Sciences, Russian Federation
Title : Investigation of bergapten’s pharmacological activity against doxorubicin induced cardiotoxicity: supporting evidence from cellular and animal models
Bijoy Krishna Banik, National Institute of Pharmaceutical Education and Research (NIPER)-Guwahati, India